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      filexlib. Pseudoporphyria LAST UPDATED: Nov 18, 2021 Introduction Refer to the chapter Porphyria Disclaimer – the author PCDS cannot accept responsibility for any misleading or incorrect statements, and the management of individual patients remains the direct responsibility of the individual doctor.
      Rofecoxib-induced pseudoporphyria. Pseudoporphyria is a photodistributed bullous eruption that usually results in superficial scarring. It has been linked to the administration of several medications including naproxen, nabumetone, amiodarone, carisprodol, furosemide, naproxen, nabumetone, other nonsteroidal anti-inflammatory drugs (NSAIDs), as
      A case series of chlorophyll-induced pseudoporphyria and proposed pathogenesis – Journal of the American Academy of Dermatology Photobiology, Phototherapy and Photosensitivity Diseases | Volume 72, ISSUE 5, SUPPLEMENT 1, AB213, May 01, 2015 A case series of chlorophyll-induced pseudoporphyria and proposed pathogenesis
      LETTER TO THE EDITOR Pseudoporphyria – a case report Editor A 69-year-old paraplegic woman with a history of antiphospholipid syndrome and vitiligo presented with tense bullae on both knees (Fig. 1). Two days previously, she had rested outdoors with flexed knees facing the sun.
      Amanita pseudoporphyria, also known as Hongo’s false death cap, is a species of agaric fungus from the genus Amanita that grows solitarily or gregarious in coniferous forests.Originally described from Japan, it is now also known in Northern India, Thailand, and Nepal. It is quite a common species in southern China and is sold in free markets, along with the similar mushroom, Amanita manginiana.
      Pseudoporphyria: A clinical and biochemical study of 20 patients | Request PDF Pseudoporphyria: A clinical and biochemical study of 20 patients Authors: Carl F Schanbacher Tufts Medical
      Erythropoietin is listed as a medication that may elicit pseudoporphyria in one article, but was suggested as treatment for pseudoporphyria in another (10, 11). There is disagreement on whether pseudoporphyria has a completely normal porphyrin profile or may have elevated plasma porphyrins, especially in the setting of renal failure (1-5, 7
      Pseudoporphyria Workup: Laboratory Studies, Procedures, Histologic Findings Drugs & Diseases > Dermatology Pseudoporphyria Workup Updated: May 11, 2018 Author: Vineet Mishra, MD, FAAD; Chief
      The various causes of pseudoporphyria include ultraviolet A exposure, chronic renal failure/dialysis and a wide range of drugs, particularly nonsteroidal anti-inflammatory drugs, diuretics, antibiotics and retinoids. Imatinib has been implicated occasionally in causing both pseudoporphyria and porphyria cutanea tarda.
      2008 •. RAM PRABAHAR. Acute intermittent prophyria (AIP) is an autosomal dominant disease that results from a defect in the enzyme porphobilinogen deaminase. Acute intermittent porphyria is the most common of hepatic porphyrias and can tax the therapeutic capabilities of the physician to the limit. Motor weakness is a major feature of an Pseudoporphyria is a disease whose symptoms mimic porphyria, with bulla on the dorsal fingers as well as milia. In contrast to porphyria cutanea tarda (PCT), pseudoporphyria is rarely associated with hypertrichosis, hyperpigmentation, or sclerodermoid change. 2-3 Pseudoporphyria is a rare entity, with less than 100 reported cases. 4-5 However
      2008 •. RAM PRABAHAR. Acute intermittent prophyria (AIP) is an autosomal dominant disease that results from a defect in the enzyme porphobilinogen deaminase. Acute intermittent porphyria is the most common of hepatic porphyrias and can tax the therapeutic capabilities of the physician to the limit. Motor weakness is a major feature of an Pseudoporphyria is a disease whose symptoms mimic porphyria, with bulla on the dorsal fingers as well as milia. In contrast to porphyria cutanea tarda (PCT), pseudoporphyria is rarely associated with hypertrichosis, hyperpigmentation, or sclerodermoid change. 2-3 Pseudoporphyria is a rare entity, with less than 100 reported cases. 4-5 However
      This is pseudoporphyria, an uncommon but well recognised side effect of non-steroidal anti-inflammatory drugs (NSAIDs), with cutaneous features in photosensitive areas indistinguishable from porphyria, but without the metabolic porphyrin abnormalities. 1 The incidence of pseudoporphyria may be as high as 10% in children taking NSAIDs for JIA. 1

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